Anatamopathological spectrum of tauopathies
Identifieur interne : 004178 ( Main/Exploration ); précédent : 004177; suivant : 004179Anatamopathological spectrum of tauopathies
Auteurs : Tamas Revesz [Royaume-Uni] ; Janice L. Holton [Royaume-Uni]Source :
- Movement Disorders [ 0885-3185 ] ; 2003-09.
Descripteurs français
- Pascal (Inist)
- Wicri :
- topic : Homme.
English descriptors
- KwdEn :
- Alzheimer disease, Anatomic pathology, Brain (pathology), Dementia (genetics), Dementia (pathology), Diagnosis, Differential, Exploration, Gerstmann Sträussler Scheinker syndrome, Human, Humans, Inclusion Bodies (pathology), Neurodegenerative Diseases (genetics), Neurodegenerative Diseases (pathology), Neuroglia (pathology), Neurons (pathology), Parkinsonian Disorders (genetics), Parkinsonian Disorders (pathology), Pick disease, Prion, Progressive, Supranuclear ophthalmoplegia, Tau protein, Tauopathies (genetics), Tauopathies (pathology), dementia, glia, neuron, parkinsonism, pathology, tau, tau Proteins (genetics), tau Proteins (metabolism).
- MESH :
- chemical , genetics : tau Proteins.
- genetics : Dementia, Neurodegenerative Diseases, Parkinsonian Disorders, Tauopathies.
- chemical , metabolism : tau Proteins.
- pathology : Brain, Dementia, Inclusion Bodies, Neurodegenerative Diseases, Neuroglia, Neurons, Parkinsonian Disorders, Tauopathies.
- Diagnosis, Differential, Humans.
Abstract
The presence of tau‐positive intraneuronal filamentous inclusions with or without additional inclusions in glial cells has been recognised as a major neuropathological feature in a significant group of neurodegenerative diseases, which are described as tauopathies. In one category of such diseases, the neuronal inclusions occur in association with extracellular deposition of a second aggregated protein (secondary tauopathies), whereas in another, the filamentous inclusions composed of tau are the sole neuropathological abnormality (primary tauopathies). Genetic studies of tauopathies in general, and in frontotemporal dementia with parkinsonism linked to chromosome 17 in particular, have significantly contributed to our knowledge about the pathogenesis not only of rare hereditary conditions but also of other more common diseases such as Alzheimer's disease and progressive supranuclear palsy. © 2003 Movement Disorder Society
Url:
DOI: 10.1002/mds.10558
Affiliations:
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Le document en format XML
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In one category of such diseases, the neuronal inclusions occur in association with extracellular deposition of a second aggregated protein (secondary tauopathies), whereas in another, the filamentous inclusions composed of tau are the sole neuropathological abnormality (primary tauopathies). Genetic studies of tauopathies in general, and in frontotemporal dementia with parkinsonism linked to chromosome 17 in particular, have significantly contributed to our knowledge about the pathogenesis not only of rare hereditary conditions but also of other more common diseases such as Alzheimer's disease and progressive supranuclear palsy. © 2003 Movement Disorder Society</div></front></TEI><affiliations><list><country><li>Royaume-Uni</li></country><region><li>Angleterre</li><li>Grand Londres</li></region><settlement><li>Londres</li></settlement></list><tree><country name="Royaume-Uni"><region name="Angleterre"><name sortKey="Revesz, Tamas" sort="Revesz, Tamas" uniqKey="Revesz T" first="Tamas" last="Revesz">Tamas Revesz</name></region><name sortKey="Holton, Janice L" sort="Holton, Janice L" uniqKey="Holton J" first="Janice L." last="Holton">Janice L. Holton</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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